J Biol Chem Collectively, our research paradigm uses a combined approach of molecular genetics, genomics and epigenomics to understand the underlying pathological and molecular basis of diabetic nephropathy. Find information and resources for current and returning patients. J Clin Invest Mitochondrial fission triggered by hyperglycemia is mediated by ROCK1 activation in podocytes and endothelial cells. This has created a paradigm shift in the field.
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Dr Darius Danesh
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Emerging Biomarkers for Risk Assessment. Clin J Am Soc Nephrol 11 The second is to understand the regulatory effects of microRNAs in microvascular complications of diabetes. This has created a paradigm daanesh in the field.
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The first is to gain insight into the pathobiology of mitochondria in the kidneys. Our personalized portal helps you refer your patients and communicate with their MD Anderson care team. Rho Kinases in diabetic nephropathy Funding Source: Pediatr Nephrol 30 5: Learn about our graduate medical education residency and fellowship opportunities.
Our laboratory has been on the forefront of identifying multiple miRNAs and their downstream effectors in the kidney. Give Now Your gift will help make a tremendous difference. Long noncoding RNA Tug1 regulates mitochondrial bioenergetics in diabetic nephropathy. We have recently published novel observations dznesh the potential effects of miRNAs in diabetic nephropathy Long J.
Choose from 12 allied health programs at School of Health Professions. Cell Metab 15 2: Deletion of scavenger receptor A protects mice from progressive nephropathy independent of lipid control during diet-induced hyperlipidemia.
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Collectively, our research paradigm uses a combined approach of molecular genetics, genomics and epigenomics to understand the underlying pathological and molecular basis adnesh diabetic nephropathy. Our group discovered that mitochondrial dynamics is a major molecular mechanism implicated in glucose-mediated microvascular organ damage Wang W et al. In particular, we want to understand the biological functions of mitochondrial dynamics and how disrupting the functions of mitochondria contribute to the pathogenesis of diabetic kidney disease.
J Biol Chem Meet Farhad Danesh, M.
Your gift will help support our mission to end cancer and make a difference in the lives of our patients. As dnaesh of our mission to eliminate cancer, MD Anderson researchers conduct hundreds of clinical trials to test new treatments for both common and rare cancers. Volunteer Change the lives of cancer patients by giving your time and talent. Strategies to reverse endothelial dysfunction in diabetic nephropathy.
J Am Soc Nephrol 27 2: MicroRNAs and their applications in kidney diseases. Nat Commun 28 7: Kidney Int 81 Learn about clinical trials at MD Anderson and search our database for open studies. J Clin Invest Mitochondrial fission triggered by hyperglycemia is mediated by ROCK1 activation in podocytes and endothelial cells. Mechanisms of protein-energy wasting in chronic kidney disease Funding Source: Donate Danfsh Giving blood and platelets provides hope for cancer patients who danrsh on the generosity of donors like you.
New insights into molecular mechanisms of diabetic kidney disease. Danesh The primary interest of our research group is to delineate the molecular mechanisms through which diabetic ranesh progresses, and to identify and characterize novel molecular targets that could potentially prevent progression of diabetic kidney disease.